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By R. Schindler (auth.), Mrs. Rosemarie Schindler (eds.)

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Additional resources for Ciclosporin in Autoimmune Diseases: 1st International Symposium, Basle, March 18–20, 1985

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Such figures argue that rheumatoid patients should be screened for this haplotype before embarking on gold treatment. In conelusion, it seems inescapable to the author that, if we successfuIly identity the genes and their products which determine susceptibility to a wide variety of autoimmune reaetions, this information wiIl profoundly affect our strategies of both prevention and therapy of autoimmunity. References 1. Basham TY, Merigan TC (1983) J Immunol 130: 1492 2. Batchelor JR, Morris PJ, Dick H, Kennedy U (1978) In: Bodmer WF, Batchelor JR, Bodmer JG, Festenstein H, Morris PJ (eds) Histoeompatibility Testing 1977.

II. Evidence for aetivation (Go to G,) signals differing in sensitivity to eyelosorine. Eur J Immunol 14:250-254 31. Koponen M, Loor F (1983) Cytoplasmie Lipid Droplets as the Possible Eventual 32. 33. 34. 35. 36. 37. 38. 39. 40. 41. 42. 43. 44. 31 Cellular Fate of Active Forms of Cyelosporin. Exp Cell Res 14912:499-512 Koponen M, Grieder A, Loor F (1984) Interferenee of Cyelosporin with Lymphoeyte Activation: Blockage of the Mitogen-Induced Increases of Lysosomal and Mitochondrial Activities. Immunology 53/ 1:55-62 Kronke M, Leonard WJ, Depper JM, Arya SK, Wong Staal F, Gallo RC (1984) Cyelosporin A Inhibits T-Cell Growth Factor Gene Expression at the Level of mRNA Transcription.

CyA treatment (months) 0 Complete remission Partial remission Failure (a) - : Negative (b) . 5 2 4 3 8 I 8 I 8 20 1 JO 2 JO 2 JO 40 40 20 40 20 20 40 20 40 20 20 I 10 I 40 •b 20 5 Autoimmunity and Cic1osporin (CyA) 36 ......... DGIoocott ,~_ 10 ",- ,~ ,. hs UndOI evA Fig. 4. Immunoreaetive insulin seeretion of in vitro cultured mouse islet eelis in the presenee of eontrol or Type I diabeties' lymphoeytes. (a) : non CyA treated diabeties in the first 6 months after onset of diabetes. (b): cic1osporin-treated diabeties.

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